Talk:CD14

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Possible role in insulin sensitivity / metabolic syndrome[edit]

"High-fat diet feeding triggers the development of obesity, inflammation, insulin resistance, type 2 diabetes and atherosclerosis by mechanisms dependent of the LPS and/or the fatty acids activation of the CD14/TLR4 receptor complex." from The Role of the Gut Microbiota in Energy Metabolism and Metabolic Disease Patrice D. Cani* and Nathalie M. Delzenne*

Current Pharmaceutical Design, 2009, 15, 1546-1558

Dunno whether this pans out or if it's just an artifact of the particular mouse model they were using, but if it does it should be worth a mention here. --24.63.27.226 (talk) 02:14, 18 March 2010 (UTC) (Dan Wylie-Sears 2, but not logged in)[reply]

Ref 12[edit]

I don't know how to edit Wikipedia refs and not worth my time, due to Wikipedia's dishonest policies. Ref 12 is incomplete. Revising it would likely trigger Wikipedia's dishonest deletionists to remove it because it is an original source stating facts directly from the source, rather than a factually incorrect second/third source. Full reference is: Funda DP1, Tucková L, Farré MA, Iwase T, Moro I, Tlaskalová-Hogenová H. CD14 is expressed and released as soluble CD14 by human intestinal epithelial cells in vitro: lipopolysaccharide activation of epithelial cells revisited. Infect Immun. 2001 Jun;69(6):3772-81. — Preceding unsigned comment added by 89.160.53.94 (talk) 07:50, 21 March 2019 (UTC)[reply]